On the issue of the development of Parkinsonian syndromes on the basis of traumatic damage to subcortical nodes

The question of the development of Parkinsonian syndromes on the basis of traumatic injuries of subcortical nodes is still far from being clarified. Some authors recognize the possibility of the development of Parkinsonian syndromes after traumatic injuries of subcortical nodes, others deny the role of trauma in the development of these symptom complexes. We do not have the opportunity to dwell on a detailed review of the literature on the issue of interest to us and we only have a few works produced in this direction. Thus, Oppenheim and Goldstein recognize trauma as a possible etiological moment in the development of Parkickson syndrome. According to Levy, even fright should be taken into account when determining the condition for the development of tremor paralysis. B and n k considers the trauma of Parkinson's sonism to be a moment that causes a latent disease to come out. In the 2 cases cited by him, he indicates that perfectly healthy men aged 31 and 42 developed trembling paralysis after injury (falling on their backs). It should be noted that one of his cases is not so much doubtful, since the patient had an alcoholic tremor before the injury. Cargo, Robert, Besanson believe that with injuries there is no reason to deny the possibility of small selective hemorrhages in the subcortical nodes, entailing the appearance of Parkinsonian syndromes. Kurt-Mendel, citing 12 cases of tremulous paralysis, recognizes trauma as an etiological moment only in cases where no other cause can be found, in addition, he also points out the need not only for predisposition, but also for a certain period between the injury and the development of the disease picture. Suk believes that with a skull injury, it is theoretically quite possible to develop Parkinsonism, but with a limb injury, the development of parkinsonia can only be as a coincidence. Russie does not attach much importance to the trauma of Parkinsonism and considers it only a stimulating moment with a hidden old disease. Gruzon, like sti Roth, believes that trauma can often be the cause of the development of Parkinson's sonism, and Meyer cites a case where a carefully collected anamnesis ruled out the possibility of developing epidemic encephalitis and where, 5 weeks after the injury, the phenomena of parkinsonism gradually developed in the left temporal and frontal regions. The author resolves the question of the pre-traumatic pre-racial situation in a negative sense. Lotmar, speaking of trauma as a causal moment of tremulous paralysis, finds it difficult to resolve the question whether in these cases the personal predisposition of the patient, the peculiarities of his constitution and blood circulation play a role, or whether the trauma activates saprophying microbes that have been in his body so far. The question also remains whether physical trauma or mental experiences play a more significant role.