REMODELING OF CARDIOVASCULAR SYSTEM AND DEVELOPMENT OF CHRONIC KIDNEY DISEASE IN PATIENTS WITH METABOLIC SYNDROME AND OBESITY: ROLE OF GENES ENOS, SUBUNIT P22-PHOX OF NADPH-OXIDASE AND MTHFR
- 作者: Saginova E1, Gallyamov M2, Balatsky A2, Kolotvin A2, Severova M1, Samokhodskaya L2, Fomin V1, Krasnova T2, Mukhin N1
-
隶属关系:
- I.M. Sechenov First Moscow State Medical University
- M.V. Lomonosov Moscow State University
- 期: 卷 84, 编号 6 (2012)
- 页面: 26-31
- 栏目: Editorial
- URL: https://journals.rcsi.science/0040-3660/article/view/31037
- ID: 31037
如何引用文章
全文:
详细
作者简介
E Saginova
I.M. Sechenov First Moscow State Medical University
Email: med02@yandex.ru
Chair of Therapy and Occupational Diseases of Medicoprophylactic Department
M Gallyamov
M.V. Lomonosov Moscow State UniversityChair of Internal Medicine of Fundamental Medicine Department
A Balatsky
M.V. Lomonosov Moscow State UniversityChair of Biochemistry and Molecular Medicine of Fundamental Medicine Department
A Kolotvin
M.V. Lomonosov Moscow State UniversityChair of Biochemistry and Molecular Medicine of Fundamental Medicine Department
M Severova
I.M. Sechenov First Moscow State Medical UniversityChair of Therapy and Occupational Diseases of Medicoprophylactic Department
L Samokhodskaya
M.V. Lomonosov Moscow State UniversityLaboratory of Gene and Cell Technology of Fundamental Medicine Department
V Fomin
I.M. Sechenov First Moscow State Medical UniversityChair of Therapy and Occupational Diseases of Medicoprophylactic Department
T Krasnova
M.V. Lomonosov Moscow State UniversityChair of Internal Medicine of Fundamental Medicine Department
N Mukhin
I.M. Sechenov First Moscow State Medical UniversityChair of Therapy and Occupational Diseases of Medicoprophylactic Department
参考
- Чазова И. Е., Мычка В. Б. Метаболический синдром. М.: Медиа Медика; 2004.
- Kerr S.M., Livingstone M.B., McCrorie T.A., Wallace J.M. Endothelial dysfunction associated with the obesity and the effect of weight loss interventions. Proc.Nutr. Soc. 2011; 70 (4): 418—425.
- Blanquicett C., Graves A., Kleinhenz D. J., Hart C. M. Attenuation of signaling and nitric oxid production following prolonged leptin exposure in human aortic endothelial cells. J. Invest. Med. 2007; 55 (7): 368—377.
- Fortuflo А., Jose G. S., Moreno M. U. et al. Phagocytic NADPH oxidase overactivity underlies oxidative stress in metabolic syndrome. Diabetes 2006; 55: 209—215.
- Комитет экспертов РМОАГ/ВНОК. Диагностика и лечение артериальной гипертензии (Рекомендации Российского медицинского общества по артериальной гипертонии и Всероссийского научного общества кардиологов). Систем. гипертензии 2010; 3: 5—26.
- Foppa M., Duncan B. B., Rohde L. E. Echocardiography-based left ventricular mass estimation. How should we define hypertrophy? Cardiovasc Ultrasound 2005; 3: 17.
- Kidney Disease Outcome Quality Initiative. Clinical practice guidelines for chronic kidney disease: evaluation, classification and stratification. Am. J. Kidney Dis. 2002; 39 (Suppl. 2): S1—S246.
- Самоходская Л. М., Балацкий А. В., Садекова О. Н., Таратина О.В. Значение генетических и средовых факторов в развитии мультифакториальных заболеваний. М.; 2011. 258—263.
- Сhen J., Muntner P., Hamm L. et al. The metabolic syndrome and chronic kidney disease in U. S. adults. Ann. Intern. Med. 2004; 140: 167—174.
- Wolf G., Hamann A., Han D. C. et al. Leptin stimulates proliferation and TGF-beta expression in renal glomerular endothelial cells: potential role in glomerulosclerosis. Kidney Int. 1999; 6 (3): 860—872.
- Bruce K.D., Byrne C.D. The metabolic syndrome: common origins of a multifactorial disorder. Postgrad. Med. J. 2009; 85 (1009): 614—621.
- He M. A., Cheng L. X., Jiang C. Z. Associations of polymorphism of P22(phox) C242T, plasma levels of vitamin E, and smoking with coronary heart disease in China. Am. Heart J. 2007; 153 (4): 640e1—640e6.
- Ueno T., Watanabe H., Fukuda N. et al. Influence of genetic polymorphisms in oxidative stress related genes and smoking on plasma MDA-LDL, soluble CD40 ligand, E-selectin and soluble ICAM1 levels in patients with coronary artery disease. Med. Sci. Monit. 2009; 15 (7): CR341—CR348.
- Ji L., Fu F., Zhang L. et al. Insulin attenuates myocardial ischemia/ reperfusion injury via reducing oxidative/nitrative stress. Am. J. Physiol. Endocrinol. Metab. 2010; 298 (4): 2416—2423.
- Dandona P., Ghanim H., Bandyopadhyay H. et al. Insulin suppresses endotoxin-induced oxidative, nitrosative, and inflammatory stress in humans. Diabetes Care 2010; 33 (11): 2416—2423.
- Zintzaras E., Papathanasiou A.A., Stefanidis I. Endothelial nitric oxide synthase gene polymorphisms and diabetic nephropathy: a HuGE review and meta-analysis. Genet. Med. 2009; 11 (10): 695—706.
- Ezzidi I., Mtiraoui N., Mohamed M. B. et al. Association of endothelial nitric oxide synthase Glu298Asp, 4b/a, and -786T>C gene variants with diabetic nephropathy. J. Diabet. Complicat. 2008; 22 (5): 331—338.
- Ahluwalia T. S., Ahuja M., Rai T. S. et al. Endothelial nitric oxide synthase gene haplotypes and diabetic nephropathy among Asian Indians. Mol. Cell. Biochem. 2008; 314 (1—2): 9—17.
- Zhu H., Wang X., Dong Y. et al. Influence of the eNOS gene on development of blood pressure and left ventricular mass: longitudinal findings in multiethnic youth. Pharmacogenet. Genom. 2005; 15 (9): 669—675.
- Lapu-Bura R. Quarshie A., Lyn D. et al. The 894T allele of endothelial nitric oxide synthase gene is related to left ventricular mass in African Americans with high-normal blood pressure. J. Natl. Med. Assoc. 2005; 97 (2): 197—205.
- Kazakov A., Müller P., Jagoda P. et al. Endothelial nitric oxide synthase of the bone marrow regulates myocardial hypertrophy, fibrosis, and angiogenesis. Cardiovasc. Res. 2011; Dec. 11. [Epub ahead of print]
- van Guldener C., Robinson K. Homocysteine and renal disease. Semin. Thromb. Hemost. 2000; 26 (3): 313—324.